Transient Ischemic Attack

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A Transient Ischemic Attack (TIA), often called a “mini-stroke”, is a temporary period of symptoms similar to those of a stroke. It occurs when there is a brief interruption in blood flow to part of the brain, spinal cord, or retina — typically lasting only a few minutes and not causing permanent damage.

Causes 

A TIA is caused by a temporary reduction or blockage of blood flow to the brain, usually due to a clot or debris in a blood vessel. Below are the most common causes and contributing factors:

1. Blood Clots (Thromboembolism)

  • Embolism from the heart: Common in people with atrial fibrillation or after a heart attack.
  • Thrombosis in cerebral arteries: A blood clot forms in a brain artery, often due to atherosclerosis (narrowing from plaque buildup).

2. Atherosclerosis

  • Fatty deposits build up in the arteries (especially carotid arteries), narrowing them and increasing the risk of clot formation.

3. Carotid Artery Disease

  • Narrowing or blockage in one or both carotid arteries (major blood vessels in the neck supplying the brain).

4. Cardiac Causes

  • Atrial fibrillation (irregular heartbeat)
  • Heart valve disease
  • Heart failure
  • Recent heart surgery
    These conditions can produce clots that travel to the brain (cardioembolic TIA).

Clinical features 

The clinical features of a TIA refer to the observable signs and patient-reported symptoms that clinicians use for diagnosis. These features are often transient, lasting less than 24 hours, and depend on which part of the brain is affected.

General Clinical Features

  • Sudden onset of symptoms
  • Short duration (minutes to a few hours)
  • Complete resolution of symptoms within 24 hours
  • No permanent neurological deficits on examination after resolution

 Neurological Features by Affected Area

Brain RegionClinical Features
Middle cerebral artery (MCA) territoryHemiparesis (face/arm > leg), hemisensory loss, aphasia (if dominant hemisphere), neglect (if non-dominant)
Posterior circulation (vertebrobasilar system)Dizziness, vertigo, diplopia (double vision), dysarthria, ataxia, bilateral weakness, drop attacks
Retinal (amaurosis fugax)Sudden, painless, transient vision loss in one eye (often described as a “curtain coming down”)
Internal carotid arteryMixed symptoms: monocular blindness, hemiparesis, hemisensory loss, aphasia

Diagnosis

  • Clinical evaluation
  • Imaging like MRI or CT scan
  • Carotid ultrasound, echocardiogram, and blood tests

Physiotherapy management 

 Although a TIA causes no lasting neurological damage, physiotherapy management is essential for:

  • Early recovery from transient impairments (e.g., balance, weakness)
  • Stroke prevention through lifestyle modification and exercise
  • Patient education and risk factor management

Assessment Phase

Before initiating treatment:

  • Detailed functional assessment:
    • Muscle strength, tone, joint mobility
    • Balance and gait (Berg Balance Scale, Tinetti)
    • Functional mobility (Timed Up and Go)
    • Cardiovascular fitness (6-minute walk test)
  • Neurological screening:
    • Cranial nerves, reflexes, coordination, sensation
  • Fall risk evaluation

Goals 

Short-Term GoalsLong-Term Goals
Improve balance and mobilityPrevent stroke recurrence
Enhance muscle strengthPromote cardiovascular fitness
Reduce fear of fallingImprove quality of life and independence
Educate on lifestyle changes

Core Physiotherapy Interventions

A. Balance & Gait Training

  • Static balance: Single-leg stance, tandem standing
  • Dynamic balance: Heel-to-toe walking, direction changes, obstacle walking
  • Gait re-education: Use of walking aids, step length & pattern correction

B. Strengthening Exercises

  • Focus on major muscle groups: quadriceps, hamstrings, glutes, core
  • Progressive resistance training (e.g., bodyweight, resistance bands, weights)
  • Frequency: 2–3 sessions/week, 10–15 reps × 2–3 sets

C. Cardiovascular Training

  • Aerobic exercise (e.g., brisk walking, cycling, treadmill)
  • Intensity: Moderate (50–70% of max HR), monitored closely
  • Duration: Start with 15–20 minutes, build up to 30–40 minutes, 3–5x/week

D. Flexibility and Postural Control

  • Stretch tight muscle groups (e.g., calves, hamstrings, hip flexors)
  • Postural exercises to correct alignment and reduce fatigue

E. Proprioception and Coordination

  • Balance boards, foam pads
  • Hand-eye and lower limb coordination drills

F. Functional Mobility Training

  • Bed mobility, sit-to-stand, chair transfers
  • Practicing real-world tasks to promote confidence

4. Patient Education

Medication adherence (e.g., antiplatelets)

Stroke warning signs (Be fast)

Importance of medical follow-up (e.g., for atrial fibrillation, hypertension)

Home exercise program and self-monitoring

Risk factor management:

Smoking cessation

Weight loss and diet

Stress management

Medication adherence (e.g., antiplatelets)

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